FARMACOS ANTIVERTIGINOSOS PDF

Shakadal Psychosocial family intervention in schizophrenia: Olanzapine versus placebo in adolescents with schizophrenia: Entre estas dos metas existe una gran diferencia. International Consensus Study of Antipsychotic Dosing. A review of its pharmacology and therapeutic potential in the treatment of schizophrenia. Curr Psychiatry Rep ;4: Cochrane Database of Systematic Reviews 2: Las madres que viven con hijos con esquizofrenia que abusan de farjacos tienen un riesgo mayor de ser agredidas. Comorbidities and Mortality in Persons with Schizophrenia: Important issues in the drug treatment of schizophrenia. Instituto Mexicano del Seguro Social.

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Diclorohidrato de betahistine Serc o betaserc-verum Este compuesto fue sintetizado en por Walter y colaboradores. Sin embargo, el mecanismo neural no se conoce muy bien. Todo depende de la dosis y de la hipersensibilidad de la persona. El resultado de una dosis oral 10 a 30 mg. Cuando su efecto cesa, se puede sentir una respuesta contraria.

Las investigaciones a fondo se han hecho en el aparato cardiovascular. La cinarazina es la 1-benzhydril-4cinnamyl piperazina. Algunos autores creen que el efecto antivertiginoso de los antagonistas del calcio se lleva a cabo gracias a sus propiedades vasodilatadoras.

Reposar en cama en el hospital o en la casa , preferiblemente en un cuarto oscuro. Aplicar una ampolla de valium de 10 mg IM, cada 12 horas. Iniciar terapia vestibular, si hay inestabilidad. Darle un bloqueador de calcio cada 12 horas. The other symptoms of desequilibrium, loss of balance, diffuse sensations of abnormal movements and pre-syncope are considered as a framework. This entity is thus a symptom, whether or not it is related to either a known or an unknown disease.

The effectiveness of its treatment depends on whether its etiology is known or unknown. For these reasons many authors have considered the pharmacological treatment of vertigo to be empirical and has discouraging results.

Drugs which interact with this symptom have been identified, thanks to animal models, in which labeyrinthic lesions have been produced, and to medicines used in kinetosis. Vertigo may have a central or peripheral origin and its treatment is aimed at blocking the action of vestibular nuclei and the vomit center.

Thus, antihistamines, calcium canal blockers, CNS stimulants, CNS depressors, antiemetics and vasodilators can all be used. It must be born in mind that, 72 hours after a peripheral lesion, there is central compensation, which occurs at the expense of the vestibular nuclei, restoring the function of the diseased peripheral organ and, finally, improving the patient.

During this period, drugs which do not interfere with compensation and control the symptoms of nausea and vomiting should be administered.

Another factor which contributes to rapid improvement is to start vestibular therapy as soon as possible. CPPB is a very precise clinical entity and its treatment must be limited to canal replacement maneuvers, without wasting time with medicines.

Kinetosis is very common in our environment and its treatment is preventive. The best medicine is scopalamine, but, because of its adverse effects, it may be replaced with kinarazine. There are a number of patients who are refractory to all therapies, both pharmacological and physical, who have been administered all types of national and international therapies, with whom we must learn to live, although uncomfortably. Key words: vertigo, neuropharmacology, anti-vertiginous, drugs, antihistamines, H1 blockers, anti-colinergic, ansiolytics, sedatives, sympathicomimetics, antiemetics, neuroleptics, blockers, calcium, vasodilators.

Correspondencia: Dr. Pedro Blanco. Hospital Universitario del Valle. Cali, Colombia. Artes Graficas Univalle.

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